In the course of Human responses to the effects of the world, we have all seen the differences in how we react to the same situation. When my last dog died I was upset for weeks, KoKo was a part of the family. After several months I ended up with two rescue bulldogs that were living under a shed, worm infested and starving. That was almost two years ago and if someone asks me about my family, Buddha and Ellie are memtioned as if I had 6 and not 4 children.
Then to other's, there animal dies and they are off to the pet store the next morning. Neither of those is an incorrect response, they just are what they are.
Was online with a beautiful and passionate woman who had lost her mother to pancreatic cancer. Tobacco has multiple cancer inducing agents that have been blamed since the 50's for this action. That is a fact- yet still we know only the partial story of what are the mutagenic agents and we are seeing 60 year smokers at 3 packs a day not succumb to cancer and 30 year olds with minor (if there is a term) tobacco use die of the disease. We are learning of genetic differences and family linkages to both extremes. So do we develop genetic markers to define can smoke? Of course not, the 80 year old died of pancreatic cancer- a disease linked to smoking.
Remember the old Eagles lyric- "We are all just prisoners here of our own device".
There is a genetic mutation in some women that greatly puts them at risk for breast cancer, whether they use tobacco or not, we recommend very young in life bilateral mastectomy because we have elucidated the gene that is present in these women.
So with Lung and Pancreatic tumours. Nicotine has a different effect on tobacco induced cancers. In a recent study observation lung cancer patients were shown to have no increase in the growth rate when NRT (Nicotine Replacement Therapy) was administered. The tumors did not change in size, rate of metastases or growth rate. This is not true of Pancreatic tumors which rapidly proliferate if the person contines nicotine exposure in any manner.
Genetics arises again. Nicotine has a a binding protein with multipleforms, some of which respond to nicotine by enhancing tumor growth. Here nicotine acts as a promoter or accelerator of tumor spread. At this point I emphasize tumor spread not tumor production.
So here is the dilemna (one of them), frame of reference. Nicotine is likely to shorten the lifespan of pancreatic cancer patients if exposure continues after thetumor developes.
The conclusion of some given half the knowledge is that nicotine is a fervent carcinogen. Tobacco smoking is the probable carcinogen and the numerous mutagenic agents within leaf tobacco smoke (and some snuffs, SNUS..). Nicotine is the unfortunate agent that acts as an accelerator in the pancrease.
So there are two findings and NRT or another form of minimal chemical contaminated nicotine used by vapors (inhaled droplet based nicotine) can be called a serious health problem or one that seems to be minimally harmful.
Pick your agenda and you have a weapon to supporrt NRT and vaping or information that shows it has a detrimental effect. It is a cyclic argument. A no real win.
My puppies, when they die will cause a different reaction in me than someone else's puppy's death. Much more later on this. Work is calling.
Then to other's, there animal dies and they are off to the pet store the next morning. Neither of those is an incorrect response, they just are what they are.
Was online with a beautiful and passionate woman who had lost her mother to pancreatic cancer. Tobacco has multiple cancer inducing agents that have been blamed since the 50's for this action. That is a fact- yet still we know only the partial story of what are the mutagenic agents and we are seeing 60 year smokers at 3 packs a day not succumb to cancer and 30 year olds with minor (if there is a term) tobacco use die of the disease. We are learning of genetic differences and family linkages to both extremes. So do we develop genetic markers to define can smoke? Of course not, the 80 year old died of pancreatic cancer- a disease linked to smoking.
Remember the old Eagles lyric- "We are all just prisoners here of our own device".
There is a genetic mutation in some women that greatly puts them at risk for breast cancer, whether they use tobacco or not, we recommend very young in life bilateral mastectomy because we have elucidated the gene that is present in these women.
So with Lung and Pancreatic tumours. Nicotine has a different effect on tobacco induced cancers. In a recent study observation lung cancer patients were shown to have no increase in the growth rate when NRT (Nicotine Replacement Therapy) was administered. The tumors did not change in size, rate of metastases or growth rate. This is not true of Pancreatic tumors which rapidly proliferate if the person contines nicotine exposure in any manner.
Genetics arises again. Nicotine has a a binding protein with multipleforms, some of which respond to nicotine by enhancing tumor growth. Here nicotine acts as a promoter or accelerator of tumor spread. At this point I emphasize tumor spread not tumor production.
So here is the dilemna (one of them), frame of reference. Nicotine is likely to shorten the lifespan of pancreatic cancer patients if exposure continues after thetumor developes.
The conclusion of some given half the knowledge is that nicotine is a fervent carcinogen. Tobacco smoking is the probable carcinogen and the numerous mutagenic agents within leaf tobacco smoke (and some snuffs, SNUS..). Nicotine is the unfortunate agent that acts as an accelerator in the pancrease.
So there are two findings and NRT or another form of minimal chemical contaminated nicotine used by vapors (inhaled droplet based nicotine) can be called a serious health problem or one that seems to be minimally harmful.
Pick your agenda and you have a weapon to supporrt NRT and vaping or information that shows it has a detrimental effect. It is a cyclic argument. A no real win.
My puppies, when they die will cause a different reaction in me than someone else's puppy's death. Much more later on this. Work is calling.
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